Search

everythinglearnresearchcommunity

for

Search:↓

Transgender patients on hormone therapy experience more acne and hair loss, but less skin inflammation.

Atopic dermatitis may have genetic causes and can be treated with pharmacologic methods, glycerin creams, and controlling Staphylococcus aureus colonization.

IL-2 treatment causes skin eruptions and other reversible side effects, and may play a role in psoriasis.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

ILC1-like cells can independently cause alopecia areata.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

IL-15 promotes hair growth and protects hair follicles.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

QLT0267 stops hair follicle cell growth and movement.

The study created special nanoparticles that effectively deliver an anti-inflammatory drug to treat skin inflammation in psoriasis.

KLF4 is important for keeping hair follicle stem cells inactive.

Activating liver X receptors can reduce fibrosis by stopping certain immune cells from releasing harmful proteins.

ILC1 cells contribute to hair loss in alopecia areata.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

miR‑339‑5p can slow down hair follicle stem cell differentiation by targeting DLX5.

Interleukin-1 increases keratin K6 production in skin cells.

The study's results on the effectiveness of low-dose IL-2 for alopecia areata and its impact on immune cells were not provided.

IL-1β inhibits human hair follicle growth.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

IL-19 and IL-24 help cells respond to DNA damage and could be targeted for cancer and age-related disease treatments.

A protein called IL-36γ causes skin side effects from certain cancer treatments when combined with a common skin bacteria.

RNase L hinders hair follicle regeneration by altering immune signals.

KLF4 is important for maintaining stem cells and has potential in cancer treatment and wound healing.

Removing the ROBO4 gene in mice reduces skin inflammation and hair loss by affecting certain inflammation pathways and gene expression.

PADI4 enzyme slows down cell growth in developing hair follicles.