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Early androgen exposure affects hormone receptor expression in adult female rats' brains, but not in males.

DHT is crucial for urethral formation, and its disruption can affect masculinization and lead to hypospadias.

Blocking androgen receptors early in life increases estrogen levels and reduces sexual motivation in male rats.

Blocking a specific enzyme in male rat fetuses leads to the development of nipples and feminized genitalia.

A 17-year-old female with adrenogenital syndrome produces very high levels of androgens, which prevent complete feminization despite high estrogen doses.

Androgens regulate Sertoli cells and affect fertility through specific receptors and coregulators.

Pesticide exposure may increase the risk of hypospadias in males.

Babies were mostly born healthy from fathers who took finasteride, but some pregnancies ended early; more research is needed.

Mouse genital development depends on male or female hormones for specific features.

EZH2 is crucial for uterine gland development and female fertility.

Finasteride treatment in male rats can reduce fertility and affect sperm development in their offspring.

Reducing 5α-reductase activity helps endometrial cells differentiate, aiding pregnancy.

Inhibiting 5α-reductase increases progesterone levels in late pregnant mares.

Paternal drug exposure can harm fertility, pregnancy outcomes, and offspring health, with specific drugs linked to various reproductive issues.

Estradiol and castration reduced prostate cancer development in rats when applied at early stages, but were ineffective after cancer was established.

Early hormones shape sex-specific differences in rat glands.

The patient had complex urinary tract issues and ambiguous genitalia, with physical features suggesting a hormonal imbalance.

Androgens may influence bladder cancer progression by affecting cellular behavior.

Exposure to fatty acids significantly increased androgen levels in female dogs, suggesting a link to conditions like insulin resistance and PCOS.

Prenatal antiandrogen treatment altered hormone levels in adult spotted hyenas, affecting males and females differently.

Androgens affect the function and health of the endometrium.

Blocking androgens in male rats increased estrogen and made them more active.

Women with a certain type of tumor had higher levels of pregnancy hormone and male hormones, which decreased after treatment.

Blocking androgens at birth reduces sexual motivation in male rats.

Male children's genital development issues can be caused by genetic mutations or environmental factors affecting hormone action.

Masculinization in affected individuals occurs gradually after puberty due to hormone changes.