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Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Thymic epithelial cells may be related to skin stem cells.

Alopecia areata involves unique activation of certain immune cells.

PCFCL may have unrecognized subtypes and needs more research.

Langerhans cells and melanocytes migrate to the skin and hair follicles during early human development.

Bone marrow-derived cells can lead to skin inflammation and tumors in mice.

Sca-1+ cells in newborn mouse skin may become fat cells.

Dual TCR Treg cells are common in mouse tissues and vary by location.

Lymphatic vessels are essential for hair follicle regeneration and growth.

Mouse touch-sensitive nerve cells adjust their connections based on competition with other similar cells.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Mouse models help target specific genes in lymphatic cells for research.

Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

People with acne have more CD4+ immune cells in their skin than healthy people.

Lymphocytes, a type of immune cell, are crucial for wound healing as they help remodel damaged areas and reduce inflammation.

Bone marrow-derived cells contribute to skin tumors, suggesting new treatment targets for non-melanoma skin cancers.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

Tph cells are linked to the severity of systemic lupus erythematosus.

Lymphatic vessels are essential for hair follicle growth and skin regeneration.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Lgr5+ stem cells do not cause skin tumors.

Researchers developed a 3D skin model with its own immune and blood vessel cells to better understand skin health and disease.

Human thymus has stem cells that can self-renew and maintain their identity.

Topical immunotherapy for alopecia areata may work by creating immune cell clusters in the skin.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.