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Three characteristics of plasmacytoid dendritic cells help tell apart lupus-related hair loss from LPP.

α3β1-integrin is crucial for maintaining normal hair follicle shape and function but not needed for the development of the surrounding skin.

Immune checkpoint inhibitors can cause skin issues but are linked to better cancer outcomes.

Two new types of eyelid cysts were identified, each with different treatment challenges.

Oncogenic K-ras causes rapid cancerous changes in the mouth's lining.

The K15 promoter effectively targets stem cells in the hair follicle bulge.

Skin cell clumping for hair growth is improved by a protein called fibronectin, which helps cells stick and move better.

Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

Stem cells in the skin help with healing and can also lead to cancer.

Outer root sheath cells consistently express certain keratins influenced by their environment.

Alopecia in patients with epidermolysis bullosa varies in severity and is often caused by skin blistering or trauma.

The early genes of a specific virus can cause abnormal skin cell growth and hair follicle changes.

Skin spheroids with both outer and inner layers are key for regrowing skin patterns and hair.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

Antiseptic particles penetrate deeper into hair follicles than non-antiseptic ones.

Murine epidermal stem cells can develop into skin structures without rejection when implanted.

Microneedling with platelet-rich plasma can cause temporary, painful lymph node swelling in some patients.

Electrical epilation damages hair follicles and surrounding skin, likely preventing hair regrowth.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

Keratin 79 marks a new group of cells that are key for creating and repairing the hair follicle's structure.

New technologies show potential for better understanding and treating skin conditions with abnormal mucin, but more research is needed for clinical use.

Removing UBE2N from skin cells causes inflammation and immune response, which can be lessened with specific inhibitors.

Dermal IgA deposition without symptoms is rare in Dermatitis herpetiformis risk groups.