Search

everythinglearnresearchcommunity

for

Search:↓

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

Increased Treg cells and IL-10 may help quick recovery in acute diffuse and total alopecia.

KIT's role in skin cells is not entirely independent, as other cells can influence its function.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Nephronectin is linked to worse breast cancer outcomes and helps cancer spread.

The nail matrix has a reduced immune response, protecting it from autoimmunity.

Non-myeloablative stem cell transplants are a promising, less toxic option for older or weaker patients, with fewer side effects and good outcomes.

QLT0267 stops hair follicle cell growth and movement.

IFN-γ production by CD4 T cells is crucial for causing alopecia areata.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

NIPP1 is important for healthy skin and could help treat skin inflammation.

Normal human melanocytes can avoid cell death through multiple pathways.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

Proinflammatory fibroblasts and vascular endothelial cells are key in keloid development.

The NUDT15 R139C variant causes thiopurine-induced leukocytopenia through a different mechanism than previously thought in Japanese patients with inflammatory bowel disease.

Long-term data and a team approach are needed to manage chronic side effects from cancer treatments.

Nail changes from immunotherapy can be managed without stopping cancer treatment.

Nilotinib may cause gray hair to return to its original color.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

WIF1 helps keep skin stem cells inactive to prevent excessive cell growth.

Regulatory γδ T cells help protect hair follicles from alopecia areata and promote hair regrowth.

Certain mice without specific receptors or mast cells don't lose hair from stress.

Autoimmune reactions may cause both alopecia areata and HAM.

Peanut agglutinin staining helps differentiate malignant melanoma from nevocellular nevus.

Special cells can help regrow hair in alopecia areata.

Fetal skin has unique immune cells different from adult skin.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

A mutation in the IKZF1 gene causes immune system overactivity, linked to autoimmune diseases like lupus.