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A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

RNase L hinders hair follicle regeneration by altering immune signals.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

ILC1-like cells can independently cause alopecia areata.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

Activating TLR3 helps improve skin and hair follicle regeneration after wounds.

A new type of nerve cell involved in itch perception was discovered.

Grasp protein helps maintain skin health after UVB exposure.

JAK inhibitors are a promising new treatment for hair loss and nail problems in alopecia areata.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

No single ideal JAK inhibitor for alopecia areata has been determined; JAK3 inhibitors may be promising with fewer side effects.

Inhibiting certain proteins harms hair follicle immunity and increases IL-33, affecting hair health.

ARQ-234, a protein designed to treat atopic dermatitis, shows increased effectiveness in early testing.

p21waf1/cip1 and p27kip1 help in hair follicle differentiation in rats.

Ruxolitinib may help treat hair loss by reducing inflammation, promoting hair growth signals, and protecting hair follicle immunity.

A genetic mutation in the hHa1 gene creates a smaller, but still functional, hair protein without causing hair problems.

A specific DNA region controls skin cell gene expression by working with certain proteins.

Genotyping for NUDT15 p.Arg139Cys can help predict thiopurine side effects in Japanese IBD patients.

Rac1 is vital for hair follicle health but not needed for skin maintenance.

Baricitinib improved platelet counts and alopecia in a 16-year-old with chronic immune thrombocytopenia.

Ritlecitinib shows promise for hair regrowth in alopecia areata patients.

IL-9 increases skin cell movement but decreases their ability to invade, and this effect is controlled by cell contractility, not by MMPs.

The protein aPKCλ is crucial for keeping hair follicle stem cells inactive and for hair growth and regeneration.

ICP5249 helps hair grow by activating a specific cell pathway.

GPRC5D is linked to the formation of hair, nails, and certain tongue areas.

CDK inhibitors may help treat ARDS and psoriasis but need more testing for safety and effectiveness.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.