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Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Mice without certain skin proteins had abnormal skin and hair development.

The Oat mouse model shows mild retinal degeneration, useful for testing treatments.

Vitamin D receptor is crucial for starting hair growth after birth.

Pinus densiflora extract may help improve memory and reduce brain inflammation.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

Prostaglandin D2 blocks new hair growth after skin injury through the Gpr44 receptor.

Different keratins have unique expression patterns in mouse skin cells.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

Transplanted whisker follicles caused hair growth on the spine of mice.

miR-195-5p reduces hair growth ability in cells by blocking a specific growth signal.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Different melanocyte types in hair follicles either survive or die during the catagen phase.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

Vaccines and targeting TrxR variants can help prevent cancer and reduce metastasis.

Overexpressing IGF-1 in mice leads to skin abnormalities and tumors.

Thyroid hormone receptors are essential for hair growth and wound healing.

TS-13 speeds up hair regrowth after chemotherapy without affecting treatment effectiveness.

Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

The hairless gene is crucial for hair health, and its mutations cause hair loss.

Increased 14-3-3 proteins may block hair cycle regression, causing hair loss.

Phosphatidylinositol 4-kinases are crucial for root growth, defense, and immunity in Arabidopsis thaliana.

Keratin2-6g is crucial for hair follicle development, with mutations causing cell degeneration and vacuolation.

Keratin 17 is important for skin's response to radiation, affecting many genes and cell division.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.