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Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

Tribulus terrestris extract significantly increases MSH expression and melanin production in mouse hair follicles.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Mice skin has components that could help with hair growth and might be used for diabetes treatment.

Collagen peptides may boost skin and hair-related gene expression.

The PP2A-B55α protein is essential for brain and skin development in embryos.

New mouse models help study melanocytic cells for melanoma research.

Increasing alkaline phosphatase in human skin cells helps to grow more hair.

A new genetic mutation in mice causes permanent hair loss and skin wrinkling.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Transplanted whisker follicles caused long hair growth on the spinal cords of mice.

Pig blood plasma can be used to promote human hair growth and is a good alternative to the commonly used serum additive.

The peptide IMT-P8 can effectively deliver proteins into the skin and cells for potential skin treatments.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Overactive Stat3 in mouse skin causes hair loss and cell structure damage.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Triptolide effectively and safely reduces actinic keratosis lesions in mice.

Scientists turned mouse skin cells into hair-inducing cells using chemicals, which could help treat hair loss.

An allele of the KRTAP13-2 gene may improve wool quality in sheep.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

KGF and its receptor are found in enlarged prostate tissue and KGF strongly increases cell growth.

Keratin-associated proteins may have roles in various mouse tissues, not just hair.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

RK81 can help promote hair growth.

Early involution in Hirosaki hairless rats' mammary glands is linked to a unique modification of STAT5A.