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Overactive Stat3 in mouse skin causes hair loss and cell structure damage.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

CD34+ cells from fat tissue help form hair follicles and blood vessels in skin.

Regulatory T cells are essential for normal and improved wound healing in mice.

The mutation helps mice handle heat better without affecting hair growth.

Recombinant thymosin β4 promotes blood vessel growth and reduces muscle damage.

HPV8 causes skin cancer by expanding specific skin stem cells.

ISCK03 stops melanin production in human melanoma cells and lightens skin color in mice and guinea pigs.

Altering SSAT affects fat metabolism and body fat in mice.

Stopping S100A3 activity slows down hair growth in mice.

Mast cells and VEGF contribute to post-surgery adhesions, and blocking VEGF can reduce these adhesions; also, certain factors affect wound healing and fetal skin heals differently with age.

Scientists found a specific group of itch-sensing nerve cells in mice important for feeling itch but not for sensing heat or touch.

A soluble factor in developing mouse whisker pads boosts nerve growth factor mRNA production.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Retinoic acid and DMSO improve gene delivery to mouse skin for potential hair and skin disease treatment.

Transplanted rat hair follicles grew hair and had increased but not fully restored nerve connections in mice.

Transgenic sheep embryos with a specific promoter were successfully created, but more research is needed for gene expression in hair follicles.

Genetically modified stem cells from human hair follicles can lower blood sugar and increase survival in diabetic mice.

Spt4 and Spt6 are essential for fat cell development.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

Skin grafts on mice can cause an immune response leading to hair loss, useful for studying human hair loss conditions.

High ODC and low K1 and K10 may indicate early skin tumors in mice.

bFGF delays hair growth in mice.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

B6.Cg-Tyr c−2J Hr hr /J mice have a stronger delayed sunburn reaction and are good for UV research.

Sdr16c5 and Sdr16c6 genes regulate a key point in lipid production that affects eye and skin gland function.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.