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NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Keratin 15 helps keep skin cells in a young, undifferentiated state.

The HPV type 11 region activates hair-specific gene expression in mice.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Type XVII collagen may help prevent skin aging.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

Mouse keratin 6 genes evolved independently from human ones and are regulated differently.

Dermal EZH2 controls skin cell development and hair growth in mice.

The hHa7 gene is regulated by androgens in certain body hair, not scalp hair.

MOF controls skin development by regulating genes for mitochondria and cilia.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

Vitamin D receptor helps control hair growth genes in skin cells.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

CAP1 decreases the expression of a hair-related protein in young Tan sheep's skin.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

Type XVII collagen helps control skin cell growth and rejuvenates skin.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.

Hair stem cells produce a protein called COL17A1 that plays a key role in their development and is linked to hair thinning and baldness.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Runx1 and CDK inhibitors help keep hair follicle stem cells inactive, affecting their future roles.

TACE/ADAM17 is essential for maintaining healthy hair and hair follicle stem cells.

The HR protein's role as a repressor is essential for controlling hair growth.

KLF4 is important for keeping hair follicle stem cells inactive.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

Keratin K14 expression varies between hair follicles and epidermis, affecting cell differentiation.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.