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Zfp157 is active in many mouse tissues during development and in specific adult cells.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Spt4 and Spt6 are essential for fat cell development.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

A Chinese man with KID syndrome had a new mutation in the GJB2 gene.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

A rabbit gene important for hair development was identified and detailed.

A specific DNA region controls skin cell gene expression by working with certain proteins.

Removing SIX1 in fat cells reduces skin fibrosis.

Keratins K6 and K16 are expressed more freely in regenerating mouse skin than K1 and K10.

S100A6 protein is linked to disease progression, especially in cancers.

Pmg-1 and Pmg-2 are new genes important for skin and mammary gland development.

IGF2BP3 gene is up-regulated in keloid patients, suggesting potential targets for treatment.

A mutation in the Sgkl gene causes defective hair growth in mice.

A rare ITGB6 gene variant causes intellectual disability, hair loss, and dental issues.

Keratin 16 delays skin maturation and affects skin and hair development in mice.

The study found nine new hair protein genes in human hair follicles.

A young Russian girl with Meier-Gorlin syndrome has two new mutations in the CDC6 gene.

Decreasing MIG6 can increase the movement and invasiveness of MEK-inhibited mutant NRAS melanoma, particularly when stimulated by EGF.

Interleukin 6 may help protect skin without causing inflammation.

GKLF/KLF4 and Sp1 control Keratin 19 gene activity, influencing cancer-related changes.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

Mutations in the KRT16 gene can cause skin and nail disorders.

Interleukin-1 increases keratin K6 production in skin cells.

The study found that variations in hair protein genes are likely due to evolutionary deletions or duplications.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Claudin 6 is crucial for normal skin and hair development.