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Scalp hair follicle cells help protect and heal skin in certain skin conditions.

IL-18 signaling helps mature Tregs move into the thymus.

EGFRi/MEKi treatments cause hair follicles to lose some immune protection, leading to inflammation.

Low m6Ascores in melanoma predict better survival and response to immunotherapy.

GLI2 activates GLI1, promoting skin tumor growth and hair development.

Hair microRNAs could be effective biomarkers for diagnosing scleroderma.

A new therapy for a skin blistering condition has not been developed yet.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

The growth of the Epstein-Barr virus in the patient's cells was linked to the worsening of her lymphoma.

Some skin growths with mucin can form hair follicles and resemble skin cancer, but a special stain can help tell them apart.

Nestin-expressing cells turn into a specific type of skin cell in hair follicles during development and in adults.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

Arg1+ macrophages may play a role in Alopecia Areata, offering new treatment targets.

Hyper-keratinisation in Meibomian glands contributes to gland dysfunction.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

PH-762 shows promise in treating skin cancer by effectively targeting and silencing PD-1 in tumors with minimal side effects.

Severe digestive issues in DRESS need early endoscopy for better treatment.

Patients with severe active alopecia areata have lower CD200 expression and an imbalance in their immune system.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

Keratins K1 and K10 are found in the inner root sheath and cuticle of human hair follicles.

A specific gene mutation in KRT86 is linked to hair disorder in a Chinese Han family.

A new skin disease in four Labrador retrievers responded well to immunosuppressive treatment.

Topical ruxolitinib quickly improves non-segmental vitiligo.

Nestin-expressing progenitor cells become outer root sheath keratinocytes.

Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

Mouse keratin 6 isoforms have different expression patterns in various tissues.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Certain biomarkers can help distinguish between irritant and allergic contact dermatitis.