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A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

SOX11 and SOX4 help skin cells act like embryonic cells to heal wounds in mice.

PRC1 is essential for proper skin development and stem cell formation by controlling gene activity.

Collagenase IV is crucial for hair growth by affecting key growth factors.

The study revealed the detailed structure of a keratin dimer, aiding understanding of how intermediate filament proteins function.

The KRT84 gene is linked to better wool quality in Gansu Alpine Fine-wool sheep.

Four antibodies were developed to help study hair follicle cell differentiation.

A girl had rickets due to a gene mutation affecting vitamin D response.

ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

The Itpr3 gene causes a specific hair pattern in mice.

CTHRC1 helps hair grow back, and plantar dermis mixture boosts it.

MOF controls skin development by regulating genes for mitochondria and cilia.

Reducing SFRP1 can promote hair growth and may help treat hair loss.

Targeting NMMHC IIA may help treat blood-brain barrier damage.

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Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.

Esrp1 is important for skin health by helping form and maintain the skin barrier.

A mutation in the KRT71 gene causes a hair disorder by disrupting hair follicle structure and texture.

KRTAP2-3 could help predict cancer recurrence by identifying specific cancer cells.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

Laminin-511 is involved in psoriasis development and can be regulated by cannabinoid receptor type 1.

Different enzymes are active in different parts of developing mouse organs.

AGD1's PH domain is essential for its role in root hair growth and polarity.

Minoxidil sulfate and pinacidil promote hair growth likely by increasing blood flow, not by activating K channels.

RAPK is a rare skin disorder with pigmented spots, mainly on hands and feet, starting in youth.

A KLK5 inhibitor effectively improved skin symptoms in a mouse model of Netherton Syndrome.

Faulty LEF1 activation causes faster skin cell differentiation in premature aging syndrome.