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Triptolide effectively and safely reduces actinic keratosis lesions in mice.

Mutations in keratin genes can cause skin and mucosa disorders.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

CAP1/Prss8 does not activate PAR2 or inhibit PN-1.

Keratinocytes can reverse the effects of the GNAQ oncogene, inhibiting melanoma cell growth.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

Certain mutations in the H6PD gene cause Cortisone Reductase Deficiency by affecting hormone production.

The keratin 5 mutation in a family with epidermolysis bullosa simplex was due to mosaicism, not a new mutation.

Androgen receptors help prostate cancer cells grow and resist drugs.

Trichohyalin helps in hair and skin cell structure and function by binding calcium and linking proteins.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

Brg1 is crucial for keeping hair follicle stem cells and repairing skin, working with the Sonic Hedgehog pathway to promote hair growth.

Basonuclin 1 (BNC1) helps skin cells multiply and move, which is crucial for wound healing.

mHa2 and mHa3 keratins have different structures and roles in mouse hair and tongue tissues.

RD26 controls root hair growth during drought by repressing key genes, helping plants save energy.

PRX01, PRX44, and PRX73 are essential for root hair growth in Arabidopsis thaliana.

ABCG2 protein marks stem-like skin cells in human epidermis.

Hair follicle cells resist turning into skin cells.

Nexin 1 may help control hair growth.

Basal cell carcinoma shows keratin patterns similar to undifferentiated hair follicle cells.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

IGF2BP3 and other m6A-related genes are linked to keloid formation and could be potential treatment targets.

Mutations in keratin genes cause cell fragility and various skin disorders.

A new genetic variant causes BRESHECK syndrome by disrupting cell growth and stress response.

CRET therapy affects inflammation in skin cells by changing cytokine levels and activating certain proteins.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.

Some hair proteins are specific to hair, while others are also found in skin cells.