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Skin and hair can regenerate after injury due to changes in gene activity, with potential links to how cancer spreads. Future research should focus on how new hair follicles form and the processes that trigger their creation.

Human skin xenografting could improve our understanding of skin development, renewal, and healing.

Lizards can regrow their tails, and studying this process helps understand scar-free healing and limb regeneration.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Different types of fibroblasts play specific roles in wound healing and cancer, which could help improve treatments.

Ginsenoside Rd may help improve skin aging by increasing collagen in the skin.

Keloid skin disorder involves abnormal fibroblast activation and immune response, linked to a group of genes including FGF11.

CD26+ fibroblasts improve skin healing and integration better than CD26− fibroblasts.

Environmental cues can change the fate and function of epithelial cells, with potential for cell therapy.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Aging reduces skin cell renewal and defense against germs due to TGFbeta, but blocking TGFbeta could help restore these functions.

Two main types of fibroblasts with unique functions and additional subtypes were identified in human skin.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

Adult fibroblasts help heart cells mature and improve heart function.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

Adult stem cells with Tp63 can form hair and skin cells when placed in new skin, showing they have hidden abilities for skin repair.

Two microRNAs affect hair follicle development in sheep by targeting specific genes.

Keratin-associated proteins have ancient origins and were used for different purposes before being adapted for hair in mammals.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Lymphatic vessels develop from various cell types and mechanisms, not just veins.

Fibroblasts, cells usually linked to tissue repair, also help regenerate various organs and their ability decreases with age. Turning adult fibroblasts back to a younger state could be a new treatment approach.

The study found new details about human hair growth and suggests that preventing a specific biological pathway could potentially treat hair graying.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Androgen is important in controlling stem cell differentiation, reducing fat development, and increasing lean mass.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Different types of long-lasting stem cells are responsible for the growth and upkeep of the mammary gland.