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LPR1 regulates root growth under low phosphate stress independently of SIZ1 in Arabidopsis thaliana.

Phospholipase C-δ1 is crucial for normal hair development.

Removing the ROBO4 gene in mice reduces skin inflammation and hair loss by affecting certain inflammation pathways and gene expression.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

Lack of TrkC receptor delays hair follicle development.

A specific mutation in the TRPV3 gene causes hair follicle cells to develop improperly, leading to hair loss.

Lack of certain fatty acids causes skin, immune, and fertility issues in mice.

Fatp4 is crucial for healthy skin development and function.

Turning off the Lhx2 gene in mouse embryos leads to slower wound healing and scars.

Par3–mInsc and Gαi3 work together to ensure proper cell division orientation in skin development.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

A gene mutation causes lanceolate hair in rats by disrupting hair shaft integrity.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

RNase L suppresses regeneration in mammals.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

ZNF750 and MPZL3 are important in causing seborrheic dermatitis.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

A missing mK6irs1 gene causes hair loss in mice.

miR-203a-3p helps hair follicle stem cells become specialized by targeting Smad1.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.

Mouse Scube3 affects teeth, tongue, vibrissae, and eye development, but not facial structure or limb growth.

Removing both KLK5 and KLK7 proteins can prevent death and skin issues in Netherton syndrome.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.