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Removing the ROBO4 gene in mice reduces skin inflammation and hair loss by affecting certain inflammation pathways and gene expression.

MPZL3 is crucial for seborrheic dermatitis development.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

PP2Acα is essential for proper hair and skin development.

Mice with a mutated Dsg3 gene showed severe symptoms but not the typical blistering of pemphigus vulgaris.

Par3–mInsc and Gαi3 work together to ensure proper cell division orientation in skin development.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

A specific mutation in the TRPV3 gene causes hair follicle cells to develop improperly, leading to hair loss.

A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

RNase L suppresses regeneration in mammals.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

Turning off the Lhx2 gene in mouse embryos leads to slower wound healing and scars.

miR-203a-3p helps hair follicle stem cells become specialized by targeting Smad1.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

Mouse Scube3 affects teeth, tongue, vibrissae, and eye development, but not facial structure or limb growth.

lncRNA2919 slows down rabbit hair growth by stopping cell growth and causing cell death.

FOXN1 is crucial for thymus development and immune response in Xenopus laevis.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

Removing both KLK5 and KLK7 proteins can prevent death and skin issues in Netherton syndrome.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

miR‑339‑5p can slow down hair follicle stem cell differentiation by targeting DLX5.

Lack of certain fatty acids causes skin, immune, and fertility issues in mice.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

ZNF750 and MPZL3 are important in causing seborrheic dermatitis.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.