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Friedreich's ataxia causes thin, weak hair with surface damage and cavities.

The Ysc84/SH3yl1 protein family is important for cell movement and the process of taking in materials by interacting with actin and cell membranes.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.

FGF-1 causes spiral ganglion neurites to branch more.

Arabidopsis Formin 2 stabilizes actin filaments, affecting cell-to-cell movement and virus susceptibility.

Trichohyalin is found in non-hair tissues and works with filaggrin in certain skin areas and conditions.

Hair follicles form hard α-keratin filaments in four steps, showing structural differences.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

Fibroblasts can be mistaken for neural cells, so functional validation is needed.

Fibronectin is essential for hair follicle regeneration by supporting stem cells.

Oxidation changes the structure of hair protein filaments, causing them to compact and rearrange.

FilaggrinHigh melanomas have active FGFR signaling and weak GNA14 and Th1 signatures.

Trichohyalin is a protein in hair follicles that helps form hair filaments.

Keratin filaments are crucial for cell structure and protection, with ongoing discoveries about their genes and functions.

Actin and alpha-smooth muscle actin help skin heal in mouse fetuses.

Pili torti hair is fragile due to loose keratin filaments and weak disulfide bonds.

Basement membrane-like ECM supports fibroblast aggregation and cohesion.

Fibroblasts, cells usually linked to tissue repair, also help regenerate various organs and their ability decreases with age. Turning adult fibroblasts back to a younger state could be a new treatment approach.

Blocking α1-integrin makes adult fibroblasts more like foetal ones, improving their movement and aiding wound healing.

Reactive lipids from aging cells change the extracellular matrix, affecting cell function and inflammation.

Mast cell and nerve fiber interactions in mouse skin change with the hair cycle.

Hard α-keratin in hair has a unique, nonordered structure, different from other fibers.

Targeting adipocyte-to-mesenchymal transition could help treat fibrosis.

The arrector pili muscle attaches to the extracellular matrix using α5β1 integrin and connects muscle cells using α1β1 integrin.

Methylene blue staining effectively highlights detailed nerve structures in rat fur.

Mast cells might contribute to hair loss by causing skin thickening.

Frontal fibrosing alopecia can be diagnosed by examining facial areas and treated with specific medications to stop its progression.