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Fibroblasts change rapidly during wound healing, forming temporary niches before stabilizing into distinct scar layers.

Reactive lipids from aging cells change the extracellular matrix, affecting cell function and inflammation.

Mast cell and nerve fiber interactions in mouse skin change with the hair cycle.

Hard α-keratin in hair has a unique, nonordered structure, different from other fibers.

Targeting adipocyte-to-mesenchymal transition could help treat fibrosis.

The arrector pili muscle attaches to the extracellular matrix using α5β1 integrin and connects muscle cells using α1β1 integrin.

Methylene blue staining effectively highlights detailed nerve structures in rat fur.

Mast cells might contribute to hair loss by causing skin thickening.

Frontal fibrosing alopecia can be diagnosed by examining facial areas and treated with specific medications to stop its progression.

Trichohyalin granules help inner root sheath cells in hair follicles harden by integrating with keratin filaments.

The hair follicle structure is more complex than thought, with new findings on protein formation.

The upper hair follicle is stable, while the lower part allows movement during hair growth.

The model helps test drugs for clubfoot fibrosis by mimicking cell environments and shows minoxidil reduces harmful collagen links.

Certain miRNAs might be involved in a hair loss condition called frontal fibrosing alopecia and could possibly help in its diagnosis.

Follicular mucinosis causes significant damage to hair follicle cells.

MC-1R in skin cells may influence inflammation and collagen production.

Imaging living plant cells shows dynamic changes in actin and endoplasmic reticulum linked to root hair growth.

Dermal fibroblasts have at least three distinct types, each with unique roles in skin structure and hair development.

Targeting immune pathways like JAK/STAT may help treat frontal fibrosing alopecia.

Keratins are crucial for cell structure, growth, and disease risk.

Targeting specific cell interactions may help treat skin fibrosis.

Oxidized trichocyte keratin has a helical dislocation in its structure.

Frontal fibrosing alopecia is linked to increased immune system activity and reduced stem cells, suggesting early treatment targeting this pathway might prevent hair follicle damage.

Macrophages and fibroblasts help repair organs after injury, affecting whether they regenerate or scar.

The cell membrane complex in mammalian hair has three distinct types with different structures and chemical properties.

Keratin structure changes during keratinization, but the exact model remains uncertain.

Skin cell clumping for hair growth is improved by a protein called fibronectin, which helps cells stick and move better.