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Stressed fibroblasts greatly increase melanin production in hair, skin, and eye cells, mainly due to a growth factor called bFGF.

The new drug delivery system improves vitiligo treatment by enhancing melanocyte activity and viability.

Finasteride reduces melanin production, possibly treating hyperpigmentation and melanoma, but needs more safety research.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

Sox9 is crucial for hair follicle stem cells to become melanocytes instead of glial cells.

PDGF-BB helps young melanocytes grow but stops mature ones from growing, and it makes melanocytes more specialized.

Human melanocytes have unique traits that affect melanoma development and prognosis.

The study found increased skin pigmentation and variable melanocyte density in a patient with Addison's disease.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.

Rhododendrol in skin-whitening products can cause skin depigmentation and immune reactions.

Human melanocytes in skin and hair follicles don't express keratin 16 or 6 naturally.

1,25-dihydroxyvitamin D3 helps melanocyte precursors from hair follicles grow and produce melanin, aiding vitiligo treatment understanding.

Tribulus terrestris extract significantly increases MSH expression and melanin production in mouse hair follicles.

cGEL hydrogel improves melanin production in skin cells, making it a promising option for skin treatments.

Overexpression of HDGF in melanocytes does not cause cancer.

A sphingolipid from human placenta may help treat vitiligo by activating melanocyte stem cells.

Rice bran extract and low-frequency electromagnetic fields together may help treat vitiligo and white hair.

Repigmentation patterns in vitiligo depend on melanocyte source, lesion status, and therapy choice.

The technique can isolate cells to help treat skin pigmentation issues.

Keratinocyte cytokines and genetic variations influence the development of moles and skin pigmentation.

Collagen membranes improve melanocyte growth for treating skin depigmentation.

SPRY1 deficiency in skin cells causes stem cells to move to the skin surface, leading to increased pigmentation.

BMI1 is essential for preventing hair greying and maintaining hair color.

The FTO gene hinders stem cells in hair follicles from becoming pigment cells.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Hair follicle-derived melanocyte transplant could effectively treat vitiligo by restoring skin color.

Stem cells can be used to create long-lasting skin cells for treating pigment disorders.

Melanocytes from human fetal hair follicles were successfully cultured, showing potential for hair disease research and clinical use.

Collagen XVII is crucial for preventing hair and pigmentation loss by maintaining melanocyte stem cells.

A human serum-enriched medium effectively supports melanocyte growth and activity without using animal-derived supplements.