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Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Only skin melanocytes, not other types, can color hair in mice.

Nanoparticles can deliver vaccines through hair follicles, triggering immune responses and providing protection.

A specific group of early-stage melanocytes is reduced in vitiligo-affected skin, which may explain treatment resistance.

A sphingolipid from human placenta may help treat vitiligo by activating melanocyte stem cells.

Melatonin may inhibit melanoma growth and has potential as a cancer therapy aid, but its effects on human skin pigmentation need more research.

Mycophenolate mofetil may safely help restore skin color in depigmentation conditions.

Hair follicle stem cells can become melanocytes to help treat skin depigmentation.

The new nanoparticles could improve melanoma treatment by working better than current options.

Peanut agglutinin staining helps differentiate malignant melanoma from nevocellular nevus.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Multiphoton microscopy helps understand and improve vitiligo treatments by visualizing skin cell changes.

A3B5 can reduce skin pigmentation and slow melanoma growth.

Merkel cell polyomavirus can infect and persist in skin cells, evading the immune system, but certain treatments can control it.

Melanocyte stem cells from non-affected skin in vitiligo patients can become functional melanocytes for potential therapy.

Apoptosis in hair follicles spreads through cell death signals, with stem cells slowing the process.

Oncogenic melanocyte stem cells can develop into melanoma similar to human cases.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Patient-derived melanocytes can potentially treat vitiligo by restoring skin pigmentation.

Fetal hair follicles have melanocytes with melanosomes at different stages, which are broken down into pigment particles in keratinocytes.

Chemotherapy reduces splenic melanin in mice.

The research found ways to activate melanocyte stem cells for potential treatment of skin depigmentation conditions.

Moles may stop growing because of cell cooperation, not just because of aging cells.

Melatonin stops melanin production after tyrosinase action, with cyclic GMP mimicking this effect.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

Retinoids or their analogs could treat skin pigmentation disorders like melasma and vitiligo.

Retinoids can help treat skin pigmentation disorders by affecting melanin production.