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Exosomes from fat-derived stem cells help repair large bone defects by attracting and enhancing bone marrow stem cells.

Corticosteroid therapy for alopecia areata can cause severe hip bone damage.

Early and late matrix progenitors in hair follicles create different cell layers, with early ones forming the companion layer and later ones forming the inner root sheath and hair shaft.

Bead-jet printing of stem cells improves muscle and hair regeneration.

Nestin-expressing progenitor cells become outer root sheath keratinocytes.

Recognizing diverse presentations of folliculotropic mycosis fungoides is crucial to avoid diagnostic errors.

Adenolipomas may develop from gland entrapment by fat tissue, showing complex and varied forms.

The study introduced a new imaging technology to track skin healing and bone marrow cell activity over time.

Mammary stem cells drive mammary gland growth by branching and cell mixing.

Scaffoldin helps form hard skin structures in chicken embryos.

Scarring alopecia involves increased immune cells and specific gene changes near damaged hair follicles.

The onychodermis helps anchor the nail bed and may aid in nail formation.

Fat, bone, and the brain are interconnected in regulating energy and health.

BMP signaling is essential for the development of touch domes.

Pilomatrixoma is a rare, harmless tumor near the eyebrow in kids, best treated with surgery.

Pigmented papules on the ear can be a rare skin condition called cutaneous amyloidosis, treatable with topical tretinoin.

Combining OMP with cyclosporine can effectively treat severe alopecia areata in children.

A rare neck cyst in a 47-year-old man showed diverse skin cell types and was not linked to HPV.

Follicular mycosis fungoides can look like a B-cell lymphoma, making diagnosis difficult.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

Milia en plaque can be caused by skin damage, tumors, or external factors.

Dendritic cells help regulate skin development and hair growth in mice.

Frontal fibrosing alopecia and ulerythema ophryogenes may be related and can evolve from one to the other.

Mutations in the POC1A gene can cause a unique form of extreme insulin resistance and short stature.

The mTurq2-Col4a1 mouse model shows that cells can divide while attached to stable basement membranes during development.

Rare ovarian tumors can cause hyperandrogenism, even if imaging appears normal.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.