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MFN2 mutations cause mitochondrial problems, unusual fat distribution, and low leptin despite high body fat.

New pyrazole-based inhibitors show promise for treating metabolic diseases and other conditions.

Mimosine is toxic to animals, causing hair loss, infertility, and growth issues, but its exact harmful mechanism is unclear.

Defective mitochondrial DNA replication causes aging symptoms and increased oxidative damage in mice.

ZDHHC13 is important for normal liver function and metabolism, affecting mitochondrial activity.

Some people have a genetic variation that makes them less effective at breaking down drugs.

Overexpressing SSAT in mice makes them highly sensitive to polyamine analogues, causing liver damage and high mortality.

Abnormal amino acid metabolism may worsen rosacea symptoms.

Mettl3 is essential for normal tissue development and self-renewal by regulating gene expression.

Early recognition of methotrexate toxicity symptoms is crucial.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Increasing isoleucine intake improved skin and health issues in an infant with maple syrup urine disease.

Food deprivation increases MST enzyme in the brain, possibly affecting energy balance.

Overexpressing SSAT enzyme reduces prostate tumor growth in mice.

The tested hair dye ingredients do not form harmful oxidized metabolites in the liver.

A baby with maple syrup urine disease improved from skin problems by adjusting his diet to correct amino acid levels.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

MFN2 mutations cause mitochondrial problems, leading to more upper body fat and lower leptin levels.

Increasing m6A levels can improve skin cell growth and wound healing.

New strategies like vitamin A and dextromethorphan can help reduce methotrexate side effects and improve patient adherence.

The 1298CC genotype in RA patients leads to higher methotrexate toxicity, while the 1298AA genotype results in better treatment response.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

The MDR1 C3435T polymorphism does not significantly affect methotrexate response in rheumatoid arthritis patients.

Targeting METTL1 may help slow papillary thyroid cancer growth and spread.

Methimazole for hyperthyroidism has known and new side effects needing more study for safety.

A new mutation in the mitochondrial DNA was found in a boy with MELAS, even though his family didn't show typical signs.

Alpha-MSH affects mitochondrial function, and MC1R mutations may increase skin aging.

Melanin may help melanoma cells grow by aiding their metabolism.