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Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Targeting TCR-Vβ2 in cutaneous T cell lymphoma shows promise for safer, more specific treatment.

Myeloid cells can turn into skin and hair cells to help heal wounds.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

B cells can both help and hinder the body's defense against melanoma.

M-CSF-stimulated myeloid cells can turn into skin cells and help heal wounds and regrow hair.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

HKN-2 antibody targets specific skin and hair cells, showing keratin complexity.

A specific type II hair keratin was identified and found in hair cortex and tongue cells.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

Mast cell and nerve fiber interactions in mouse skin change with the hair cycle.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Hair follicle cells help protect against immune attacks by regulating T-cell activity.

Schwann cell and M2 macrophage interactions contribute to keloid growth by increasing matrix deposition.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Merkel cell polyomavirus can infect and persist in skin cells, evading the immune system, but certain treatments can control it.

MC-1R is present in skin cells and may help reduce inflammation.

Skin cancer often starts from Lgr5+ progenitor cells.

Cells that move well may improve hair loss treatments by entering hair follicles.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

Type 2 immunity helps control mite growth in hair follicles, preventing damage.

Skin cells control immune cell placement, helping the skin respond better to challenges.