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A new genetic model may improve treatment and diagnosis for certain inherited skin diseases.

A new method can detect mutations in mice by observing changes in hair follicle cells.

The improved genome of the African spiny mouse helps study its tissue regeneration.

A Gsdma3 mutation causes hair loss due to stem cell damage from skin inflammation.

PP2Acα is essential for proper hair and skin development.

A new genetic mutation was found causing hair and eye issues in a boy.

Defective mitochondrial DNA replication causes aging symptoms and increased oxidative damage in mice.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

CDP is crucial for lung and hair follicle cell development.

The Itpr3 gene causes a specific hair pattern in mice.

A new genetic mutation in the CDH3 gene causes hair loss and eye problems in young people.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

LncRNA MSTRG.14227.1 hinders hair follicle development in cashmere goats, affecting cashmere quality.

Multiphoton microscopy effectively images mouse skin layers and structures.

Higher miR-34a levels and the A variant of the MIR-34A gene are linked to increased risk and severity of alopecia areata.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

Hair root analysis can effectively detect somatic mosaicism in double cortex syndrome.

A specific pathway involving AR, miR-221, and IGF-1 plays a key role in causing common hair loss.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

MicroRNA-302 helps improve the conversion of body cells into stem cells by blocking NR2F2.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

Lack of Fgf21 slows hair growth by affecting gene interactions.

PRC1 is essential for proper skin development and stem cell formation by controlling gene activity.

Phospholipase C-δ1 is crucial for normal hair development.

JMJD3 and NF-κB activate Notch1, which is essential for skin cell movement and wound healing.

Targeting MIG and MCP-1 may help treat inflammation in alopecia areata.

Mutations in Gasdermin A3 cause skin inflammation and hair loss by disrupting mitochondria.

Agouti and AGRP affect pigmentation and obesity, with implications for metabolic disorders.

The study concludes that a genetic mutation in TFM mice leads to reduced androgen receptor activity, affecting the body's response to male hormones.