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Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Certain proteins help nerve cells branch, and other findings relate to cancer, stem cell behavior, and cell division.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

Matriptase is highly active in hair follicles and sebaceous glands, especially during hair growth phases.

Scientists can control how skin stem cells divide by using different treatments.

Dermal cell activity increases during hair growth in rats.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

DNA flow cytometry effectively evaluates how different conditions affect hair growth cell activity.

Proteolytic activity in mouse skin changes with hair cycle stages, peaking in early anagen.

The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.

Minoxidil boosts hair cell growth, while cyclosporin does not.

Steroid sex hormones activate matriptase in prostate cancer cells but not in breast cancer cells.

L-cystine, D-pantothenat, and miliacin together significantly boost keratinocyte growth and metabolism.

XMU-MP-1 stops cell growth in a human mini-organ and reduces the effectiveness of the chemotherapy drug paclitaxel.

Melatonin affects cell growth differently based on its concentration.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

SSAT is a key enzyme affecting cell growth and metabolism, with potential but risky use in disease treatment.

Minoxidil affects cell growth in two ways: low doses increase growth, while high doses slow it down and can be toxic.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

Matriptase imbalance contributes to cancer development and spread.

Matriptase is crucial for skin barrier, hair growth, and may contribute to skin cancer.

Oligomeric procyanidins may help hair grow by affecting cell growth and the hair growth cycle.

Suppressing ODC activity reduces tumor growth in hair follicles.

Activin B helps start and grow hair follicles in mice.

Topical putrescine and spermine increased DNA synthesis in hairless mouse skin.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

Biotin helps maintain hair follicle growth by affecting cell death and survival signals.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.