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A new method effectively predicts estrogen-related health effects for early screening.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Diphenyl cresyl phosphate has low toxicity but can harm the liver, kidneys, adrenal glands, and testicles at high doses.

New inhibitors may reduce gut toxicity from cancer drugs.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

The assay effectively detects hormonal activity of certain chemicals.

Some HIV treatments can harm mitochondria, potentially causing side effects like lactic acidosis and nerve damage.

A mouse gene mutation increases the risk of skin cancer.

The 1298CC genotype in RA patients leads to higher methotrexate toxicity, while the 1298AA genotype results in better treatment response.

These chemicals can be toxic, causing health issues like cancer, liver damage, and irritation.

Compound 1 showed promising anticancer activity.

Certain pesticides and fungicides can interfere with male hormone functions, potentially causing reproductive issues in male rats.

The risk of skin tumors becoming malignant depends on the specific skin cell type affected.

Oncogenic K-ras causes rapid cancerous changes in the mouth's lining.

Androgen receptor inactivation delays skin cancer onset and reduces incidence in mice, with gender differences in susceptibility.

Low levels of tenuazonic acid can severely damage vital organs.

The study developed a tool to predict how gut microbes process foods and drugs, showing that similar compounds often share metabolic pathways and effects.

TCDD changes skin cell growth and keratin production in mice.

Budesonide and N-acetylcysteine reduced tumors and alopecia in mice, regardless of FHIT gene status.

Alpha-MSH affects mitochondrial function, and MC1R mutations may increase skin aging.

The hair dye formulations tested did not cause long-term toxicity or cancer in mice.

C.I. Acid Orange 3 caused cancer in female rats but not in male rats or mice.

Lower dosages of 2-(difluoromethyl)-dl-ornithine might reduce side effects, supporting further research.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

Dimethylarsinic acid speeds up skin tumor growth in certain mice.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Gamma radiation enhances the antioxidant properties of Hibiscus rosa-sinensis extract without causing toxicity.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

Maternal exposure to artificial food coloring may increase skin disease risk in rat offspring.