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Topical rapamycin may effectively treat fibrous papules on the face.

The malignant pilomatricoma showed strong epithelial keratin expression, suggesting it may not calcify.

mTOR inhibitors may help treat lung fibrosis.

Adipocytes can change into fibroblast-like cells to help with wound healing.

EF and PXE not closely related.

Canine dermal papilla cells and fibroblasts have distinct growth patterns and protein expressions.

FGF-1 causes spiral ganglion neurites to branch more.

Reactive lipids from aging cells change the extracellular matrix, affecting cell function and inflammation.

The conclusion is that fat tissue in the skin is a new finding in Frontal fibrosing alopecia and may contribute to hair follicle and muscle degeneration.

A rare benign tumor with hair follicle features was found on a man's trunk.

Corticosteroids can reduce scarring in acne keloidalis by targeting specific cells.

Tissue stiffness is influenced by contractility, which suppresses collagen breakdown.

Researchers identified specific genes that are important for mouse skin cell development and healing.

UV exposure harms skin by causing fibroblast loss, but T cells help fibroblast survival.

Krox20 overexpression in fibroblasts may play a role in abnormal scar formation and could be a target for new treatments.

Multi-peptide factors from fibroblasts may stimulate hair growth by increasing growth factors and β-catenin in hair cells.

Three genes, BMP4, POSTN, and WNT5A, may help treat keloids.

Pilomatrixomas likely originate from the hair matrix due to changes in hair keratin expression.

Targeting Hedgehog signaling may help treat ligamentum flavum fibrosis.

The hamartoma is an abnormal hair growth with limited development in the upper hair follicle.

The skin can still regenerate and function well even with fewer fibroblasts.

Alopecia areata may involve disrupted mesenchymal function in hair follicles.

A specific molecular switch, driven by MAPK/ERK signaling, helps spiny mice heal wounds by regenerating skin instead of forming scars.

Schwann cell and M2 macrophage interactions contribute to keloid growth by increasing matrix deposition.

Combining anti-androgenic, anti-inflammatory, and anti-fibrotic treatments may improve hair loss outcomes.

Ossification in trichilemmal cysts is more common than previously believed.