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Overexpressing PKCepsilon in mice reduces papillomas but increases carcinomas.

CREB, a protein that can promote cancer traits, is controlled by β-catenin in skin cancer cells.

hsa_circ_0002980 can help stop liver cancer cells from growing and spreading.

A woman with Kallman's syndrome also developed alopecia universalis, a condition not previously linked to Kallman's.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

FOL-026 peptide can help repair blood vessels and promote growth, offering potential treatment for vascular diseases.

Keratin 18 helps diagnose and predict cancer progression and affects cancer growth and spread.

Non-coding RNAs may be key in diagnosing and treating rare skin disorders.

Key genes linked to hair growth and cancer were identified in hairless mice.

Wnt and Shh signaling are key in noggin-induced tumors, and blocking them can slow tumor growth.

New cancer drugs can cause skin side effects like rashes, dry skin, hair changes, and nail problems.

Ca²⁺-mediated protein citrullination controls cell growth in the CNS and may help treat brain tumors.

Senescent fibroblasts can help start tumors in normal skin cells.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

Mutations in the ACTB gene cause Becker’s nevi and may lead to muscle issues in Becker’s nevus syndrome.

HPV16-transformed cells can change human skin cell properties, aiding tumor growth.

Androgen receptor overexpression can increase prostate cancer cell growth even without hormones.

Eliminating senescent cells can prevent and reverse chemotherapy-induced peripheral neuropathy.

Deleting TNFα gene reduces skin cancer risk in certain mice.

Truncated hHb1 keratin may play a role in breast cancer cell transformation.

Human hair follicle cells can be turned into neural stem cell-like cells, which might help treat brain diseases.

Zebrafish need MYC and FGF to regenerate inner ear hair cells.

Vav2 changes how hair follicle stem cells' genes work as they age, which might improve regeneration but also raise cancer risk.

Targeting multiple pathways and understanding genetic mutations are crucial for effective melanoma therapy.

mTOR may link different pathways in hair follicle tumor formation.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

A new benign nail tumor called onychoblastoma was identified.