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Alopecia areata may involve stress-related changes affecting hormone receptors, leading to reduced cortisol production.

RNase L hinders hair follicle regeneration by altering immune signals.

Neurosteroids help protect fetal brains from asphyxia damage.

Vitiligo is caused by the immune system attacking skin pigment cells.

Microglia are essential for zebrafish brain repair by controlling inflammation.

Finasteride reduces inflammation and improves immune response after trauma by altering hormone levels.

Removing certain hair follicle stem cells worsens skin reactions to allergens.

A thorough, team-based approach and clear communication improve outcomes in complex neuropsychiatric lupus cases.

Botulinum toxin type A reduces itch and inflammation from histamine and may help treat certain skin conditions and hair loss.

Mild exercise reduces pain in older rats through a brain chemical, while intense exercise reduces pain in all rats through a different pain-blocking process.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

Early NAS level changes affect alcohol consumption vulnerability.

Hair follicle stem cell exosomes help nerve regeneration.

Ethanol withdrawal reduces the brain's response to certain calming steroids, making seizures harder to control.

Methylene blue staining effectively reveals detailed nerve structures in rat snouts.

Age-related immune changes significantly affect disease development in other systems.

Lung immune cells can be trained to better fight infections after a virus.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Immune cells help keep skin healthy and repair it, but imbalance can cause disease.

Inflammation helps stem cells repair tissue by directing their behavior.

Stress turns hair white by depleting color-giving cells in hair follicles through a specific neurotransmitter related to the body's stress response.

Corticosteroid treatment reduces inflammation and alters hair keratins in alopecia areata.

miR-214-3p helps nerve repair and recovery.

The article concludes that better understanding gene regulation related to seasonal changes can offer insights into the mechanisms of seasonal timing in mammals.

Contact immunotherapy can change immune responses in alopecia areata, suggesting new treatment targets.

Meis2 is essential for touch sensation and nerve function in mice.

Alopecia areata involves immune system imbalances that may lead to depression and anxiety.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.