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NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Keratin K6irs is a marker for the inner root sheath of hair follicles in mice and humans.

Four specific keratins in hair follicles help understand hair structure and function.

A new protein, mK6irs, is found in specific hair layers and may help understand hair growth and diseases.

KRT2.13 gene is similar to KRT2.9 but not expressed in hair follicles.

New compounds may help treat heart disease by activating specific potassium channels.

The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

K25, K27, and K28 are found in all inner root sheath layers of hair, while K26 is only in the cuticle.

Inward rectifier potassium channels are important in many body functions and diseases, and could be potential drug targets.

KRT72 gene helps form hair.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

A mutation in the IKZF1 gene causes immune system overactivity, linked to autoimmune diseases like lupus.

Trichosanthes kirilowii extract helps wounds heal faster.

Lack of KSR1 stops certain skin tumors in mice.

A new keratin, hK6irs1, is found in all layers of the hair follicle's inner root sheath.

ACKR2 helps prevent skin scarring and hair loss by controlling inflammation.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

Irisin can promote hair growth by activating a specific signaling pathway.

Certain genetic changes in the KRT82 gene may cause patchy skin in New Zealand rabbits.

Too much IKZF1 and Ikaros protein may cause alopecia areata.

Targeted siRNA therapy may be a promising treatment for KID syndrome by reducing mutant gene expression and improving cell communication.

A variant in the KRT31 gene causes a rare hereditary hair disorder called monilethrix.

Integrin-linked kinase is crucial for normal skin healing.

Certain KIR genes in Indian SLE patients are linked to disease severity and could be biomarkers.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.