Search

everythinglearnresearchcommunity

for

Search:↓

Chemotherapy-induced hair loss is partly due to decreased laminin-511 and increased laminin-332.

RSPO1 could help create new diabetes treatments by increasing pancreatic β cells.

The NF-κB effector p65/RelA activates hair keratin genes, aiding hair formation.

Human hair keratins K85 and K35 create unique filament patterns important for early hair formation.

Truncated LTBP-1 disrupts TGF-β signaling, affecting hair growth.

A peptide from hair follicle stem cells promotes hair growth by activating specific skin cells.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

Protease Nexin-1 is found in human hair growth cells and is affected by male hormones.

BMP signaling prevents hair growth by stopping stem cell activation.

Elf5 controls skin cell growth and development, making it a potential target for skin treatments.

MicroRNA-181a slows sheep hair growth by targeting GNAI2 and affecting a key growth pathway.

MicroRNA-302 helps improve the conversion of body cells into stem cells by blocking NR2F2.

p53 protein may help protect or kill neurons under stress.

CXXC5 can both suppress and promote cancer, making it a complex target for treatment.

JMJD3 and NF-κB activate Notch1, which is essential for skin cell movement and wound healing.

pCLCA2 protein may help maintain skin structure and function.

Lack of TrkC receptor delays hair follicle development.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

Mouse skin has special cells in the epidermis that decrease with age and are linked to keratinocyte stem cells.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Overexpression of certain genes can shorten hair by disrupting the hair-growth cycle.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

A specific genetic marker is linked to male pattern baldness in Han Chinese men.

NFATc1 controls hair stem cell activity, affecting hair growth and could be a target for hair loss treatments.

The C-allele and CC-genotype in the PTPN22 gene lower the risk of alopecia areata.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

The SNP rs6457452 is linked to a higher risk of alopecia areata in Koreans.

CCL 27 levels are similar in people with and without alopecia areata.