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JAK inhibitors may effectively treat lichen planopilaris and frontal fibrosing alopecia with minimal side effects.

Lichen planopilaris involves disrupted fat metabolism, increased scarring, and mast cell activity.

Not having the gene PLAAT3 leads to fat loss, high insulin resistance, and abnormal fat levels in the blood due to a disruption in fat cell development and function.

Bendamustine combined with tucidinostat may effectively treat adult T-cell leukemia.

A new engineered treatment shows promise in curing heart fibrosis.

Clq deficiency is linked to systemic lupus erythematosus symptoms.

Protein-losing enteropathy worsened with lupus symptoms but improved with prednisolone.

Ca²⁺-mediated protein citrullination controls cell growth in the CNS and may help treat brain tumors.

Keratin 14 is uniquely found in a specific group of placental cells.

New biomarkers and potential treatments for skin diseases were identified.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Being allergic to linalool, a common fragrance ingredient, might contribute to developing frontal fibrosing alopecia.

Injectable platelet-rich fibrin is better than platelet-rich plasma for promoting hair growth.

Impaired LEF1 activation speeds up skin cell development in Hutchinson-Gilford Progeria Syndrome.

FGFR2 signaling controls Merkel cell formation in different skin regions.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

Taking L-tryptophan supplements might cause a condition similar to scleroderma in some people, which can get better after stopping the supplement and starting corticosteroid therapy.

Mouse skin cells can become sperm-like cells in the lab.

Newly designed proteins can effectively degrade specific proteins in cells, offering a potential new therapy method.

Amotosalen-treated donor T-cells can prevent late CMV infection after bone marrow transplants.

Removing the ROBO4 gene in mice reduces skin inflammation and hair loss by affecting certain inflammation pathways and gene expression.

T cells and inflammation are crucial in atherosclerosis, with anti-inflammatory treatments showing promise.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Too much IKZF1 and Ikaros protein may cause alopecia areata.

Trps1 is essential for proper hair follicle development.

Donor lymphocyte infusions effectively treated leukemia relapse but caused vitiligo and alopecia areata.

The early genes of a specific virus can cause abnormal skin cell growth and hair follicle changes.

Profilin1 speeds up wound healing.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.