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Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

RACB in barley is crucial for cell polarity and nucleus positioning, aiding fungal infection.

The research found specific genes and proteins that affect how fast chickens' feathers grow, which is not solely determined by traditional inheritance patterns.

Tudor-SN is important for immune cells, and polyamines can promote hair growth.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

CEP135 may predict cancer outcomes, and targeting PLK1 could help treat certain sarcomas.

The circadian clock in skin cells controls their growth and rest cycles.

Polyamines are crucial for skin tumor development, and inhibiting them can prevent tumors.

Whn is essential for hair growth, and its malfunction causes hair loss.

Gap junctions help control feather pattern formation by enabling cell communication.

CUX1 boosts sheep hair cell growth and affects curl patterns.

BMI1 is essential for preventing hair greying and maintaining hair color.

Hair follicles in mutant mice self-organize into ordered patterns within a week.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

Mouse and human skin development share similar fibroblast timelines.

GKLF/KLF4 and Sp1 control Keratin 19 gene activity, influencing cancer-related changes.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Key skin cell regulators and gene organization changes are crucial for skin cell development and could help treat skin disorders.

The genes OVOL1 and OVOL2 are important for hair growth and may be involved in a type of skin tumor.

Deleting MED1 in skin cells causes hair loss and skin changes.

Genes related to keratin, skin cell differentiation, and immune functions are key in hedgehog skin and spine development.

The Foxn1(-/-) phenotype disrupts hair growth and affects skin stem cells.