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Exposure to certain chemicals before birth and during childhood may affect puberty timing.

Neonatal hair can help determine drug exposure during pregnancy.

Prenatal pesticide exposure may cause earlier breast development in girls.

PFAS exposure in adolescence may delay puberty in females.

EPI-NCSCs from hair follicles may help treat brain development issues in mice.

Lower 2D:4D finger ratios in HS patients suggest prenatal hormone influence on the disease.

TRα and CRABPII genes change their activity levels during goat fetal skin development.

Southern elephant seals develop hair follicles and skin layers before birth, with moulting starting a week after birth and finishing in three weeks.

Higher prenatal anxiety is linked to lower cortisol levels in newborns' hair.

Immune cells are essential for early hair and skin development and healing.

Treatment during development affects hormone balance and sexual behavior in male rats.

miR-29a-5p prevents the formation of early hair structures by targeting a gene important for hair growth and is regulated by a complex network involving lncRNA627.1.

Prenatal retinoic acid exposure did not affect mouse vibrissal follicle development.

Developing human skin has immune cells with memory-like features.

Prenatal and postnatal environments both affect PCOS development and gut microbiota in mice.

Pre-natal, internal, and external factors may contribute to the development of Autism Spectrum Disorder (ASD).

3D cultivation and prenatal stem cell exosomes improve stem cell treatment results, especially for hair loss and age-related issues.

Red Sokoto goat fetuses develop firmer, pigmented skin with hair follicles as they grow.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

Mutations in the LSS gene cause hair loss and may affect brain development, with varying severity.

Higher early lead exposure is linked to delayed puberty in girls.

Early exposure to fluoride, lead, and certain nutrients affects puberty differently in boys and girls.

High levels of androgens during early development may cause PCOS-like symptoms.

Women with polycystic ovary syndrome tend to have a longer anogenital distance.

Prenatal THC exposure may harm ovarian health and fertility.

Higher anxiety during pregnancy is linked to lower cortisol levels in newborns' hair.

Exposure to certain glycol ethers during pregnancy may be linked to lower verbal comprehension in children.

Understanding embryologic layers improves skin disorder diagnosis and supports developing targeted therapies.

Longer anogenital distance may indicate a higher chance of having polycystic ovary syndrome, and measuring this distance along with hormone levels could improve diagnosis.

Androgen receptors in the brain affect metabolism and reproduction differently in males and females, and may help manage PCOS symptoms in females.