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High doses of testosterone disrupt hormone levels and receptor expression in the uterus, affecting fertility.

Leydig cell tumors in the ovary can cause high testosterone and male traits in postmenopausal women but are treatable with surgery.

Rare ovarian tumors can cause increased male hormones in postmenopausal women; surgery is an effective treatment.

Male children's genital development issues can be caused by genetic mutations or environmental factors affecting hormone action.

Early hormones shape sex-specific differences in rat glands.

A woman's severe male hormone excess was caused by a small, hard-to-find ovarian tumor.

A woman's small ovarian tumor causing high androgen levels was missed by several scans but found during surgery.

A woman's excess male hormone symptoms were caused by a rare benign tumor in her ovary.

A special blood test helped find the overactive ovary in a teen with PCOS, and removing that ovary improved her condition.

Finasteride slows down motherly behavior in first-time pregnant rats.

A 31-year-old woman was diagnosed with Complete Androgen Insensitivity Syndrome much later than usual, leading to a call for earlier detection and treatment guidelines.

Surgery improved symptoms and normalized hormone levels in a woman with an adrenal tumor.

Testosterone treatment effectively causes male physical development in transgender male adolescents but may lead to side effects like acne, higher BMI and blood pressure, lower good cholesterol, and decreased bone density.

The document concludes that antiandrogenic drugs like cyproterone acetate and spironolactone are effective but not permanent treatments for skin-related androgenization in women.

Testosterone makes the connections in the uterus lining simpler and lowers certain protein levels, which might lead to infertility.

Pregnancy-related hormone changes affect Y1 receptor gene expression in mice.

Rare ovarian tumors can cause hyperandrogenism, even if imaging appears normal.

A new genetic mutation causes severe Leydig cell hypoplasia, affecting sexual development.

Blocking androgens at birth reduces sexual motivation in male rats.

The enzyme 5α-reductase type 1 is important for blood vessel development and fertility in the uterus.

Surgical removal of a rare ovarian tumor improved symptoms and hormone levels in a postmenopausal woman.

Cyproterone acetate is a safe treatment that causes modest feminization in transgender female adolescents, and works better with added estrogens.

A thorough diagnostic process and teamwork are crucial for managing complex hyperandrogenism in postmenopausal women.

Hirsutism is excessive male-pattern hair growth in women, often treated cosmetically or with hormone therapy if contraception is also desired.

Hyperthyroidism can hide signs of high androgen levels in females.

The discovery of 5α-reductase deficiency in guevedoces led to the development of important urologic medications.

Genetic risk for PCOS can affect children's growth, metabolism, and development from early life into adulthood.

Lack or blocking of SRD5a, a key component in hormone creation, can lead to conditions like pseudohermaphrodism and affect hair growth, bone mass, muscle strength, and reproductive health. More research is needed on its regulation from fertilization to adulthood.