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Hearing decline in SAMP8 mice starts before outer hair cell loss and may be linked to other changes.

Vitamin D receptor is crucial for starting hair growth after birth.

Foxn1 is essential for hair pigmentation by directing pigment transfer to hair cells.

Hoxc8 gene helps start mammary gland development by controlling specific signals.

A new genetic mutation linked to CARASIL syndrome and small artery disease was found in a Chinese family.

Pro-IGF-II improves muscle repair in old mice.

Mutant Cx43 causes slower wound healing and hair growth issues in ODDD.

CD44 variant changes start alopecia areata, but don't maintain it.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

Hedgehog signaling controls hair follicle development and can affect skin cancer growth.

CD61 is important for mouse tooth cell growth and works through Lgr5.

Rothmund-Thomson syndrome type 2 can cause chronic, poorly healing wounds.

Double-stranded RNA helps regenerate hair follicles by increasing retinoic acid production and signaling.

PPARγ is essential for maintaining healthy skin, controlling inflammation, and ensuring proper skin barrier function.

Alopecia areata can be triggered by specific immune cells without genetic or environmental factors.

Mutations in the Whn gene affect hair keratin gene expression differently.

The hairless gene is crucial for hair health, and its mutations cause hair loss.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Mutations in the P2RY5 gene cause autosomal recessive woolly hair.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

PrPC is important for neural differentiation in cattle and mouse embryonic stem cells.

HDAC1 is crucial for skin development and preventing tumors.

LncRNA MSTRG.14227.1 hinders hair follicle development in cashmere goats, affecting cashmere quality.

SOX2 helps reduce wound size and pressure ulcer formation by suppressing oxidative stress and increasing antioxidant activity in mice.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

Raptor and Rictor have stable expression in hair cycles, with Raptor marking stem cells and Rictor involved in hair shaft formation.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

HoxC genes are crucial for normal hair and nail development.