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ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

The condition is linked to chromosome 12, but no mutations were found in the known genes.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

Retinoids help treat psoriasis effectively, but they can cause side effects and need more research on safe use with other treatments.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

The treatment cleared psoriasis in some patients but caused side effects in most.

RXRa is crucial for Th2 immune cell development and may link nutrition to immune health.

Certain enzymes react strongly with some hormones in rat skin during hair growth, mainly in sebaceous glands and hair sheaths.

New retinoids are effective for various skin conditions and are being developed to have fewer side effects.

DPR can show different hair characteristics, as seen in two brothers with normal hair.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

A girl had rickets due to a gene mutation affecting vitamin D response.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

The document's conclusion cannot be provided because the document is not available or cannot be read.

The enzymes Aldh1a2 and Aldh1a3 are involved in making retinoic acid in hair follicles and have different roles in hair growth.

Dermatopathia pigmentosa reticularis causes skin discoloration, hair loss, and nail problems.

Retinoids are effective for severe acne and psoriasis but can cause serious side effects and birth defects.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

High doses of certain vitamins can cause serious side effects and health risks.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe side effects.

Prenatal retinoic acid exposure increased cell proliferation in mouse hair follicles without affecting their development.

A vitamin D receptor mutation causes rickets and affects immune responses.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.