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A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

Hhip-Cre effectively targets dermal papilla cells for gene manipulation in hair biology.

Ritlecitinib significantly improves scalp hair regrowth in alopecia areata patients over time.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Retinoic acid reduces cell damage from oxidative stress.

Adalimumab significantly improves quality of life for patients with moderate to severe hidradenitis suppurativa.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

RXR and RAR proteins in skin may help with cell growth, hair growth, and gland function.

Growth hormone therapy can improve symptoms and growth in children with Vitamin-D Dependent Rickets Type-2.

Giving dihydrotachysterol to mother rats caused skin hardening and bone issues in their babies through milk.

Samcyprone ointment is effective for treating common warts if a sensitization reaction occurs first.

KRT14 gene variants cause dermatopathia pigmentosa reticularis, affecting nails, teeth, and hair.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

Measuring minimal residual disease on day 15 helps identify high-risk leukemia patients.

Young HS patients often have other physical and mental health issues, and research on HS covers a wide range of topics including genetics, triggers, treatments, and the need for more data.

Using testosterone-stimulated weanling rats can effectively replace castrated rats for anti-androgen testing, reducing animal stress.

Patients prefer the higher 50 mg dose of ritlecitinib for better hair regrowth despite higher risks.

The protein RSL4 is crucial for making root hairs longer by controlling genes related to cell growth.

RORA affects hair follicle stem cells' structure and movement, potentially helping treat hair loss.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

The assay effectively detects hormonal activity of certain chemicals.

A patient with Rhupus was diagnosed with Rowell syndrome and treated with various medications.

Cinacalcet may help treat hereditary vitamin D resistant rickets safely.

Rhupus is a complex syndrome that combines rheumatoid arthritis and lupus, making diagnosis challenging.

RXR-alpha is strongly expressed in both normal and psoriatic skin and may help in skin cell differentiation and hair growth.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.