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Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

Basal cell carcinomas have much higher levels of Vitamin D3 receptors compared to healthy skin.

RXR and RAR proteins in skin may help with cell growth, hair growth, and gland function.

RXR agonists may promote hair growth in humans.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

A single amino acid change in the vitamin D receptor can disrupt its function and lead to hair loss.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.

RXR-alpha is strongly expressed in both normal and psoriatic skin and may help in skin cell differentiation and hair growth.

Vitamin D receptor mutations can cause alopecia by affecting hair growth genes.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

The vitamin D receptor can work without its usual activating molecule.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Retinoids or their analogs could treat skin pigmentation disorders like melasma and vitiligo.

Alitretinoin may help treat alopecia areata.

α-Mangostin can naturally lighten skin by reducing melanin.

Increased PPARGC1α relates to hair thinning in common baldness.

Retinoids can help treat skin pigmentation disorders by affecting melanin production.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

The rs1128977 gene variant may affect cholesterol and body measurements.

LGD1069 effectively prevents breast tumors in mice without toxicity.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Newer retinoid drugs are effective for skin conditions but have significant side effects.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Nuclear hormone receptors play a significant role in skin wound healing and could lead to better treatment methods.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

The mineralocorticoid receptor may play a role in skin and hair health and could be a new target for treating related disorders.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.