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Transgenic sheep embryos with a specific promoter were successfully created, but more research is needed for gene expression in hair follicles.

New SRD5A2 variants may disrupt protein function, aiding diagnosis and treatment of related disorders.

Human induced pluripotent stem cells can be used to create cells that help grow hair.

Mice with a virus similar to COVID-19 had skin damage, but a special treatment helped repair it.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Activating β-catenin can turn skin cells into hair follicles.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

The research identified and described a gene important for hormone conversion in endangered catfish, which varies in activity during different reproductive stages and after hormone treatment.

Cat color patterns are determined early in development by gene expression and epidermal changes, with the Dickkopf 4 gene playing a crucial role.

Mice with enhanced regeneration abilities may help develop new regenerative medicine therapies.

Rodent models helped understand psoriasis but none perfectly replicated the disease.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

Altering SSAT affects fat metabolism and body fat in mice.

CCCA in women of African ancestry may be caused by PADI3 gene mutations and intense hair grooming.

ATP6AP1‐CDG can cause hearing loss, hair loss, and protein issues, even in female carriers.

The review suggested the study needed to clarify its purpose, compare with non-immortalized cells, and provide more details on methods.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Vitamin D3-activated cell byproduct promotes hair growth in mice by increasing blood vessel growth.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

New mutations in the EBP gene cause CDPX2, affecting bones, skin, eyes, and hair, with females generally less affected than males.

The document concludes that mouse models are helpful but have limitations for skin wound healing research, and suggests using larger animals and genetically modified mice for better human application.

Blocking the JAK/STAT pathway may help reduce skin sensitivity in Xeroderma pigmentosum.

Dendritic cells help regulate skin development and hair growth in mice.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

The mutation causing Hutchinson-Gilford progeria syndrome leads to severe skin problems and early death in mice.

Recombinant human TSG-6 speeds up wound healing in diabetic mice.

Activating β-catenin in certain skin cells speeds up hair growth in mice.

Nrf-2-modified stem cells from hair follicles significantly improve ulcerative colitis in rats.