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Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

TGF-β2 helps activate hair follicle stem cells by counteracting BMP signals.

Rapamycin may help treat Leigh syndrome by targeting protein kinase C.

Tcf3 and Lef1 are key in deciding skin stem cell roles.

Modified KGF mRNA helps skin cells grow and move faster, which may improve wound healing.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

New cancer treatments show promise in reducing tumor growth and improving skin regeneration in mice.

Lack of TrkC receptor delays hair follicle development.

Canine hair follicles have stem cells in the bulge region.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

SGK3 is essential for proper hair growth and health.

Mcl-1 can activate Wnt signaling in skin cells, promoting growth and possibly cancer.

CXCL12+ fibroblasts help recruit neutrophils to fight skin infections.

ERK activation spreads between cells, influencing cell division and wound healing.

Radiation treatment causes skin fibrosis by increasing certain fibroblast subpopulations, but using a c-Jun inhibitor or fat grafting can reduce this effect.

Cholecystokinin may help reduce skin inflammation in psoriasis.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Foxn1 helps skin cells mature by controlling a specific protein's activity.

Protein Kinase C shows promise for cancer treatment, but more research is needed to develop effective inhibitors.

Blocking mTORC1 reduces skin tumor growth in mice.

Wnt/β-catenin signaling is crucial for cell fusion in placental development.

Erlotinib causes skin inflammation through IL-1, which can be reduced by anakinra.

EGF–FGF2 helps mouse stem cells grow and become more like nerve cells.

Human basal cell carcinomas and squamous cell carcinomas have unique gene expression patterns not fully mirrored in mouse models.

Csdc2 helps hair growth in cashmere goats by regulating specific genes.

A protein called IL-36γ causes skin side effects from certain cancer treatments when combined with a common skin bacteria.

JAK/STAT1 pathway causes hair loss during chemotherapy by reducing Shh in hair follicles.

Epidermal β-catenin activation changes the dermis by signaling different fibroblast types.

Researchers found that hair follicle stem cells can become squamous cell carcinoma due to Ras activation, which could lead to new treatments.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.