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Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

Parental uveitis causes hair loss in offspring of C57BL/6J mice.

Isoallopregnanolone may be a safe and effective treatment for reducing tics in a mouse model of Tourette syndrome.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

Raccoon dogs in Schleswig-Holstein, Germany, were found with sarcoptic mange, showing severe skin issues and potential for spreading the disease.

Topical ceramide treatment partially improves the skin condition in Jack Russell Terriers with a genetic skin disorder.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

A new mRNA variant of the SCF gene in sheep skin produces a shorter, different protein.

Lack of Evi in skin causes psoriasis-like symptoms in mice.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Targeting ornithine decarboxylase can help prevent skin cancer.

Dexamethasone speeds up hair loss in mice, while cyclosporin A slows it down.

Baricitinib treatment helped reduce hair loss symptoms in mice by decreasing inflammation-related immune cells.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

The spiny mouse regenerates ear tissue asymmetrically, with gene expression differences possibly explaining its unique healing abilities.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Smad2 and Smad3 are essential for normal skin development, and their absence causes severe skin issues and cancer.

Oncogenic melanocyte stem cells can develop into melanoma similar to human cases.

The enzyme DHHC13 is essential for healthy hair and skin, and its deficiency leads to hair loss and skin problems.

The spiny mouse can regenerate its skin without scarring, which could help us learn how to heal human skin better.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Skin cells can naturally limit the growth of cancerous changes by balancing cell renewal and differentiation.

Increased skin pigmentation in mice reduces bioluminescent signal accuracy.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

The Agouti gene influences pigmentation and may have a developmental role in deer mice.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Injecting alpha-melanocyte-stimulating hormone in mice improved skin healing and reduced scarring.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.