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TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

The 736T>A mutation in the LIPH gene is common in Japanese people with autosomal recessive woolly hair.

An 8-year-old with severe hair loss regrew all hair after six months of tofacitinib treatment.

Microneedle arrays deliver botulinum toxin effectively for sweat suppression, similar to injections.

LGD1069 effectively prevents breast tumors in mice without toxicity.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

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Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

New compounds were made that block an enzyme linked to breast cancer better than existing treatments.

Tissue-engineered skin substitutes can model junctional epidermolysis bullosa and may help develop gene therapy.

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The QuantAnts machines can find cancer markers and create CRISPR targets for them.

A new TP63 mutation was found in a baby with EEC syndrome, showing the need for TREC testing to check for immune issues.

The KAP8-1 gene affects skin, hair follicle development, and wool quality in different sheep varieties.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

CT-707 is effective and safe for treating certain Chinese lung cancer patients.

Tofacitinib significantly improved hair loss and lesions in a patient with overlapping autoimmune disorders.

Experts discussed treatments for skin conditions in children, emphasizing hydration, cautious medication use, and early intervention for infections.

Mutations in the TSPEAR gene cause a new form of ectodermal dysplasia affecting hair and tooth development.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

The boy's severe skin disorder is caused by two new mutations in his TGM1 gene.