Search

everythinglearnresearchcommunity

for

Search:↓

The SYP123-VAMP727 complex is important for transporting materials that harden the root hair shank in Arabidopsis.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Lack of TrkC receptor delays hair follicle development.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

A mutation in the KRT71 gene causes a hair disorder by disrupting hair follicle structure and texture.

NM2 and RLC phosphorylation are essential for normal inner ear hair cell function.

A new genetic mutation in the hairless gene causes a rare hair loss disorder.

Overexpression of COX-2 causes early hair loss in mice, but can be prevented with a COX-2 inhibitor.

Sostdc1 controls the size and number of hair and mammary gland structures.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

CTIP2 may help in skin development and maintenance.

Hairless mammals evolved quickly in both gene and non-gene areas related to skin and hair.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Certain genetic changes in the KRT82 gene may cause patchy skin in New Zealand rabbits.

Fgfr2b helps maintain healthy skin and prevent cancer.

Overactive Wnt5a disrupts hair follicle orientation in mice.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

The gene Msx2 is crucial for hair follicle regeneration during wound healing.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Deleting Crif1 in mouse skin disrupts skin balance and hair growth.

The angora mouse mutation causes long hair and hair defects due to a gene deletion.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Normal cells outcompete and remove mutant cells in the pancreas with the help of the EphA2 receptor.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.