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Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

New targets for making and using brain-synthesized steroids could lead to better treatments for brain disorders and alcoholism.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

Pregnancy increases certain GABA_A receptors in rat brains, influenced by steroid levels.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Hairless protein can block vitamin D activation in skin cells.

Short-chain fatty acids from *Cutibacterium acnes* cause skin inflammation, contributing to acne.

Adenosine may promote hair growth by increasing FGF-7 levels in dermal papilla cells.

FOXN1 gene variants cause low T cells and immune issues from birth.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

A mother's western diet can make her milk toxic, causing inflammation and hair loss in babies.

Increasing calcium sensing receptor speeds up skin and hair development in mice.

FER and TOR signaling help root hair growth in low temperature and low nitrate conditions.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

ΔNp63α helps control a protein that stops cancer cells from spreading.

Melatonin helps Cashmere goats grow more hair by affecting certain genes and cell pathways.

Late pregnancy helps repair brain damage in rats due to the GABAergic system.

EP 2 receptor is essential for heart repair by helping macrophages work properly.

Wnt3a activates certain genes in hair follicle cells, including a newly discovered one, EP2, which may affect hair growth.

TIP39 and PTH2R help control calcium levels and skin cell development.

Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Vitamin D boosts a specific protein in skin cells linked to hair follicles.

Endothelial TLR2 is crucial for timely wound healing, but HFSC TLR2 is not needed.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Cholesterol transport in hair follicles decreases from growth to regression phase.

A substance called 15-deoxy prostaglandin J2 can cause hair follicle cells to die, which might explain how prostaglandin D2 can lead to hair loss.

Glypican-1 is important for blood vessel growth in hair follicles and could help treat hair loss.