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Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

RCS-01 cell therapy is safe and improves skin gene expression.

NFATc1 controls hair stem cell activity, affecting hair growth and could be a target for hair loss treatments.

Cellular senescence has both cancer-blocking and cancer-promoting effects, and targeting senescent cells may improve health and lifespan.

Ceramide Synthase 4 is essential for normal hair growth and preventing hair loss.

PRC1 is essential for proper skin development and stem cell formation by controlling gene activity.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Acid inside cells speeds up aging and turns on aging signs in mice.

Ceramide Synthase 4 is crucial for maintaining hair follicle stem cells and preventing hair loss.

Apoptosis, not oxidative stress, is linked to aging in mice with mtDNA mutations.

Smad4 is crucial for muscle repair, especially in aging, by promoting cell growth over differentiation.

The Siah1 and Siah2 genes are active in mouse skin development and hair growth, especially right after birth.

Aging skin cells change their lipid profiles due to stress, affecting skin health.

TAZ boosts fat cell formation in goat stem cells by activating a specific signaling pathway.

SARMs show promise but need more evidence to prove they're better than traditional androgens.

Sesn2 protects inner ear hair cells from damage by regulating certain cell survival pathways.

Environmental factors affect reproductive traits by altering the SRD5A1 gene.

Sphingosine 1-phosphate helps control mechanical pain.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Manipulating 5α-reductase type 2 can affect liver fat production and glucocorticoid effects.

Blocking the Mitochondrial Pyruvate Carrier causes stress in hair follicles, which can be reduced by an ISR inhibitor.

ARL15 is important for fat cell development and the release of the hormone adiponectin.

Targeting THY1 can improve skin repair and healing.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

TCF/Lef1 is essential for skin barrier function by regulating lipid metabolism.

Blocking 5α-reductase can reduce sleep deprivation-related behavioral issues in rats.