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The April 2014 issue of "Fertility and Sterility" discussed various reproductive health topics, including hormone therapy benefits, sperm and genetic factors in male infertility, and the link between PCOS and diabetes.

Spermidine supplements can help extend the hair growth phase and may be useful for treating hair loss.

Men who start puberty later may have lower sperm quality and different hormone levels in adulthood.

Finasteride effectively reduced priapism episodes in children with sickle cell disease without side effects.

Lycopene protects against testicular damage caused by finasteride.

Finasteride can cause long-lasting sexual dysfunction even after stopping the drug.

Finasteride 1-mg doesn't harm sperm or pregnancy chances.

Finasteride use in male rats may harm their male offspring's liver function.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Ultrasound shows 96% of young men with erectile dysfunction after using finasteride have abnormal penile tissue.

5α-Reductase inhibitors can negatively affect male sexual function and, in some cases, significantly reduce sperm count, but these effects may be reversible.

Finasteride for hair loss can cause erectile dysfunction due to tissue fibrosis.

A 12-year-old boy with PAIS successfully developed male characteristics using high-dose testosterone and anastrozole.

Reproductive gynecologists are the main referrers for male infertility evaluations in North America.

5α-reductase deficiency can cause ambiguous genitalia and gender dysphoria, treatable with testosterone.

The document does not determine if adults with aphallia are fertile.

Finasteride reduces blood vessel formation in seminal vesicles.

Older men have different fertility histories and factors affecting fertility, suggesting they may benefit from earlier fertility evaluations.

A spermidine-based supplement may help hair grow longer by keeping it in the growth phase.

Individualized treatment and support can help most couples with recurrent implantation failure achieve pregnancy.

Finasteride doesn't change rat testicle weight or sperm production.

Blocking androgens at birth reduces sexual motivation in male rats.

Finasteride causes reproductive changes in male rats.

The combination of cyproterone acetate and testosterone enanthate is highly effective in preventing sperm production and could be a good reversible male contraceptive.

Finasteride affects frog testes by increasing testosterone, decreasing 5α-DHT, and impacting genes related to reproduction and other functions.

A certain mutation in the fetal alpha 5-reductase gene is linked to a higher risk of late miscarriage.

Finasteride in male rats causes liver and metabolic issues in their offspring.