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Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

RNA aptamers can specifically block FGF5-related cell growth, potentially treating related diseases or hair disorders.

Two specific genetic markers increase the risk of hair loss in Asian populations.

LSD1 is essential for healthy skin development and creating the skin's protective barrier.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

PAK4 is crucial in cancer progression, brain development, and could be a therapeutic target, especially through the PAK4-CREB axis.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

IGN genes may regulate hair growth and could be targeted for hair-loss treatments.

The study found that p63 needs signals from morphogens to help skin cells differentiate properly.

Hereditary hyperplastic gingivitis in silver foxes may be linked to errors in the MAPK signaling pathway, influenced by androgens.

Sox13 is a new marker for early hair follicle development and differentiation.

Key genes are crucial for Drosophila wing development and could be insecticide targets.

Spexin levels are lower in PCOS patients and linked to metabolic and hormonal issues.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

A new mouse mutation causes skin and hair defects due to a gene change.

Genes linked to wool fineness in sheep have been identified.

Arab APS1 patients have unique and recurrent AIRE gene mutations.

HPV8 causes hair follicle stem cells to grow, leading to skin lesions.

Mutations in the Whn gene affect hair keratin gene expression differently.

Certain genetic variants in ERN1, TACR3, and SPPL2C are linked to when Alzheimer's disease starts.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

A common mutation in the hHb6 gene is linked to monilethrix, but other factors may also play a role.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

A new genetic mutation in the TRPS1 gene causes Trichorhinophalangeal Syndrome, leading to specific hair, dental, and bone issues.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.