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Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

New compounds may help treat prostate issues without affecting androgen receptors.

5 Alpha-Reductase Inhibitors reduce the effects of testosterone by blocking its conversion into a stronger form.

Increasing SSAT makes skin more prone to cancer.

EPC-K is stable except at very acidic pH or when exposed to sunlight, and it can decompose in low ethanol concentrations.

Targeting ornithine decarboxylase can help prevent skin cancer.

Giving dihydrotachysterol to mother rats caused skin hardening and bone issues in their babies through milk.

Certain genetic mutations can lower bad cholesterol and reduce heart disease risk, leading to effective cholesterol-lowering drugs.

Higher levels of certain proteins may increase or decrease rosacea risk.

Amino acid storage proteins are essential for maintaining stem cells in female fruit flies.

Sphingosine 1-phosphate affects inflammation and gene expression in different aorta cells.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

Overexpressing SSAT enzyme reduces prostate tumor growth in mice.

Human sebaceous glands can grow normally for a week without certain growth factors, and adding estrogen reduces their oil production without affecting cell growth.

Inhibiting ALOX12 can help hair cuticle maturation by increasing S100A3 citrullination.

Testosterone affects androgen receptors and lipid storage in cells, while DHEA does not convert to testosterone or affect these receptors in the same way.

A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.

Cow milk sugars increase fat production and inflammation in skin oil cells.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Simvastatin helps hair follicle stem cells turn into skin cells.

A defective protein in progeria causes cell death and atherosclerosis, but a treatment targeting cell stress may reduce these effects.

Lower CD200R1 on certain immune cells is linked to more severe rheumatoid arthritis and immune imbalance.

Dihydrotestosterone increases the activity of an enzyme in pubic skin cells that converts testosterone to dihydrotestosterone.

Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

Long-term oral and topical treatments improved skin condition in a goldendoodle with a genetic disorder.

3α-AdiolG is a good marker for androgen activity in women with excessive hair growth and decreases with anti-androgen treatment.

A new ARID1B gene variation causes Coffin-Siris syndrome 1 and early high myopia in a Chinese family.

Four genes are potential markers for hair loss condition alopecia areata, linked to a specific type of cell death.

Dsg1 is essential for maintaining a healthy skin barrier in mice.