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Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

A woman with lupus and Kikuchi-Fujimoto disease improved with treatment.

The research found that specific stem cells maintain skin gland openings and that disrupting their activity can cause blockages or early cancer signs, indicating a need for targeted treatments.

MMP-19 may worsen skin diseases by affecting skin growth and inflammation.

Hair transplants can trigger a rare scarring hair loss that is hard to treat.

YAP1 is important for skin regeneration and may affect skin disorder treatments.

Familial dyskeratotic comedones are a rare, inherited skin condition that is hard to treat but may improve slightly with topical retinoids and urea cream.

Antibodies help identify glycoproteins in normal skin and tumor cells.

Fos protein is crucial for cell transition to cornification in keratinized tissues.

The material combining eggshell protein and scaffold helps wounds heal faster and regenerates tissue effectively.

The cat's skin condition was resistant to treatment and did not improve.

Trichodysplasia spinulosa can occur after a heart transplant due to immunosuppressive drugs.

Sebaceous glands can form new hair follicles when activated, but hair follicle bulges cannot.

Lef1 is essential for normal skin, hair growth, and healing wounds in mice.

Adalimumab improved folliculitis decalvans in most patients.

Cutaneous lupus erythematosus is a chronic skin disease that can progress to systemic lupus in some cases and requires treatment to prevent recurrences and scarring.

DP and DS cells are different from DF cells in structure and function.

CI-1033 causes skin lesions in rats, similar to humans, due to EGF receptor inhibition.

Fibroblast Growth Factor-9 helps repair heart damage after a heart attack by creating new blood vessels, especially in diabetics.

Skin inflammation can worsen intestinal inflammation and colitis.

Lymphocytes may hinder hair stem cells, causing hair loss without scarring.

ES cell-derived stem cells can help regenerate skin and form gland-like structures.

Isotretinoin reduced inflammation in tufted hair folliculitis, but hair tufting remained.

Ephrins slow down skin and hair follicle cell growth.

Mast cells likely promote skin scarring and fibrosis, but their exact role is still unclear.

A woman's progressive hair loss was correctly diagnosed as a rare condition called fibrosing alopecia in a pattern distribution after initially being mistaken for a more common type.

Targeting non-Smad pathways in TGF-β signaling may improve keloid treatment.